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DNA Repair and Human Disease / by Adayabalam S. Balajee.

Por: Colaborador(es): Tipo de material: TextoTextoSeries Medical Intelligence UnitEditor: Boston, MA : Springer US, 2006Descripción: xii, 150 páginas, recurso en líneaTipo de contenido:
  • texto
Tipo de medio:
  • computadora
Tipo de portador:
  • recurso en línea
ISBN:
  • 9780387368023
Formatos físicos adicionales: Edición impresa:: Sin títuloClasificación LoC:
  • RB155-155.8
Recursos en línea:
Contenidos:
Human Premature Aging Disorders and Dysfunction of DNA Repair -- DNA Repair Aspects for RecQ Helicase Disorders -- Trichothiodystrophy -- Roles of the BRCA1 and BRCA2 Breast Cancer Susceptibility Proteins in DNA Repair -- Radiosensitivity of Cells Derived from Down Syndrome Patients -- The Fanconi Anemia/BRCA Pathway -- Is Ataxia Telangiectasia a Result of Impaired Coordination between DNA Repair and Cell Cycle Checkpoint Regulators? -- Mechanisms of DNA Damage and Repair in Alzheimer Disease -- Orchestration of Telomeres and DNA Repair Factors in Mammalian Cells -- Defective Solar Protection in Xeroderma Pigmentosum and Cockayne Syndrome Patients.
Resumen: DNA Repair and Human Disease highlights the molecular complexities of a few well-known human hereditary disorders that arise due to perturbations in the fidelity of diverse DNA repair machineries.
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Springer eBooks

Human Premature Aging Disorders and Dysfunction of DNA Repair -- DNA Repair Aspects for RecQ Helicase Disorders -- Trichothiodystrophy -- Roles of the BRCA1 and BRCA2 Breast Cancer Susceptibility Proteins in DNA Repair -- Radiosensitivity of Cells Derived from Down Syndrome Patients -- The Fanconi Anemia/BRCA Pathway -- Is Ataxia Telangiectasia a Result of Impaired Coordination between DNA Repair and Cell Cycle Checkpoint Regulators? -- Mechanisms of DNA Damage and Repair in Alzheimer Disease -- Orchestration of Telomeres and DNA Repair Factors in Mammalian Cells -- Defective Solar Protection in Xeroderma Pigmentosum and Cockayne Syndrome Patients.

DNA Repair and Human Disease highlights the molecular complexities of a few well-known human hereditary disorders that arise due to perturbations in the fidelity of diverse DNA repair machineries.

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