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001			289611
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007			cr nn 008mamaa
008			150903s2009 xxu| o |||| 0|eng d
020			_a9781607613114 _99781607613114
024	7		_a10.1007/9781607613114 _2doi
035			_avtls000343335
039		9	_a201509030846 _bVLOAD _c201405050255 _dVLOAD _y201402061153 _zstaff
040			_aMX-SnUAN _bspa _cMX-SnUAN _erda
050		4	_aRC321-580
100	1		_aMcCandless, David W. _eautor _9303385
245	1	0	_aThiamine Deficiency and Associated Clinical Disorders / _cby David W. McCandless.
250			_a1.
264		1	_aTotowa, NJ : _bHumana Press, _c2009.
300			_brecurso en línea.
336			_atexto _btxt _2rdacontent
337			_acomputadora _bc _2rdamedia
338			_arecurso en línea _bcr _2rdacarrier
347			_aarchivo de texto _bPDF _2rda
490	0		_aContemporary Clinical Neuroscience
500			_aSpringer eBooks
505	0		_aEarly Chemistry -- Early Thiamine Deficiency -- Thiamine Deficiency in Mammals -- Beriberi -- Wernicke’s Disease -- Central Pontine Myelinolysis, Alcoholic Cerebellar Degeneration, and Marchiafava–Bignami Disease -- Leigh’s Disease -- African Seasonal Ataxia -- Inherited Ataxias -- Thiamine Deficiency in Serious Illness -- World Health Concerns -- Epilogue: Future Prospects.
520			_aThe past twenty years have seen remarkable advances in neuroscience, neurology, imaging techniques, and diagnostic strategies. These advances have been successfully applied to many different diseases, including thiamine deficiency and associated clinical disorders. Syndromes such as beriberi, Wernicke’s disease, Leigh’s disease, African Seasonal Ataxia, and various inherited ataxias, have all benefited from improved scientific approaches. Thiamine Deficiency and Associated Clinical Disorders represents an intriguing area of both basic and clinical investigation. Modern imaging and diagnostic strategies have facilitated the rapid treatment, and potential reversal of these clinical disorders. The fusion of laboratory and clinical knowledge serve as an example of how research can translate to successful treatment. This book is designed to bring together cogent results from both basic and clinical investigation. These data will be of interest to neurologists, internists, nutritionists, biochemists, neurochemists, neuroscientists, and many others with interest in thiamine deficiency. About the Author: Dr. David W. McCandless is the John J. Sheinin Professor of Anatomy in the Department of Cell Biology and Anatomy at The Chicago Medical School, Rosalind Franklin University, North Chicago, IL, USA. He has over 35 years of laboratory research into basic mechanisms of various metabolic encephalopathies. He serves as Editor-in Chief of the journal Metabolic Brain Disease (Springer), now in its 25th year. Dr. McCandless has been on the faculty or research staff at The University of Vermont College of Medicine, NIH-NINCDS, University of Texas Medical School at Houston, and The Chicago Medical School, and was a visiting professor at Washington University School of Medicine. Dr. McCandless has published in journals such as The J. Clinical Investigation, Nature, Proc. National Academy of Sciences, Amer. J. of Physiology, Brain Research, J. Neurochemistry, Teratology, Epilepsia, Stroke, and many others.
590			_aPara consulta fuera de la UANL se requiere clave de acceso remoto.
710	2		_aSpringerLink (Servicio en línea) _9299170
776	0	8	_iEdición impresa: _z9781607613107
856	4	0	_uhttp://remoto.dgb.uanl.mx/login?url=http://dx.doi.org/10.1007/978-1-60761-311-4 _zConectar a Springer E-Books (Para consulta externa se requiere previa autentificación en Biblioteca Digital UANL)
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